國家衛生研究院 NHRI:作者相關文件
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    "Lay, JD"的相關文件 

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    顯示 12 項.

    類別 日期 題名 關聯 檔案
    [莊雙恩] 期刊論文 2024-07-01 The role of AKR1B10 in lung cancer malignancy induced by sublethal doses of chemotherapeutic drugs -
    [莊雙恩] 期刊論文 2023-10-04 Intervention of AXL in EGFR signaling via phosphorylation and stabilization of MIG6 in non-small cell Lung cancer -
    [莊雙恩] 期刊論文 2019-11 PTBP1-mediated regulation of AXL mRNA stability plays a role in lung tumorigenesis -
    [莊雙恩] 期刊論文 2016-02 Negative feedback regulation of AXL by miR-34a modulates apoptosis in lung cancer cells -
    [莊雙恩] 期刊論文 2014-02 Oxidative stress enhances AXL-mediated cell migration through an AKT1/Rac1-dependent mechanism [Erratum:Free Radical Biology and Medicine. 2013 Dec;65:1246-1256.] -
    [莊雙恩] 期刊論文 2013-12 Oxidative stress enhances Axl-mediated cell migration through an Akt1/Rac1-dependent mechanism -
    [莊雙恩] 期刊論文 2008-09-18 Receptor tyrosine kinase AXL is induced by chemotherapy drugs and overexpression of AXL confers drug resistance in acute myeloid leukemia -
    [莊雙恩] 期刊論文 2007-04-15 Sulfasalazine suppresses drug resistance and invasiveness of lung adenocarcinoma cells expressing AXL -
    [蘇益仁(2002-2015)] 期刊論文 2007-09 Pathogenesis and mechanism of disease progression from hemophagocytic lymphohistiocytosis to Epstein-Barr virus-associated T-cell lymphoma: Nuclear factor-kappa B pathway as a potential therapeutic target -
    [蘇益仁(2002-2015)] 期刊論文 2007-05 Epstein-Barr virus (EBV) latent membrane protein-1 down-regulates tumor necrosis factor-alpha (TNF-alpha) receptor-1 and confers resistance to TNF-alpha-induced apoptosis in T cells - Implication for the progression to T-cell lymphoma in EBV-associated hemophagocytic syndrome -
    [蘇益仁(2002-2015)] 期刊論文 2005-11-01 Epstein-Barr virus LMP1 inhibits the expression of SAP gene and upregulates Th1 cytokines in the pathogenesis of hemophagocytic syndrome -
    [蘇益仁(2002-2015)] 期刊論文 2003-01 Epstein-Barr virus latent membrane protein-1 mediates upregulation of tumor necrosis factor-alpha in EBV-infected T cells: Implications for the pathogenesis of hemophagocytic syndrome -

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