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    Please use this identifier to cite or link to this item: http://ir.nhri.org.tw/handle/3990099045/15355


    Title: Disease-modifying therapies and features linked to treatment response in type 1 diabetes prevention: A systematic review
    Authors: Felton, JL;Griffin, KJ;Oram, RA;Speake, C;Long, SA;Onengut-Gumuscu, S;Rich, SS;Monaco, GSF;Evans-Molina, C;DiMeglio, LA;Ismail, HM;Steck, AK;Dabelea, D;Johnson, RK;Urazbayeva, M;Gitelman, S;Wentworth, JM;Redondo, MJ;Sims, EK;Tobias, DK;Merino, J;Ahmad, A;Aiken, C;Benham, JL;Bodhini, D;Clark, AL;Colclough, K;Corcoy, R;Cromer, SJ;Duan, D;Felton, JL;Francis, EC;Gillard, P;Gingras, V;Gaillard, R;Haider, E;Hughes, A;Ikle, JM;Jacobsen, LM;Kahkoska, AR;Kettunen, JLT;Kreienkamp, RJ;Lim, L-L;Männistö, JME;Massey, R;McLennan, N-M;Miller, RG;Morieri, ML;Most, J;Naylor, RN;Ozkan, B;Patel, KA;Pilla, SJ;Prystupa, K;Raghavan, S;Rooney, MR;Schön, M;Semnani-Azad, Z;Sevilla-Gonzalez, M;Svalastoga, P;Takele, WW;Tam, CH-t;Thuesen, ACB;Tosur, M;Wallace, AS;Wang, CC;Wong, JJ;Yamamoto, JM;Young, K;Amouyal, C;Andersen, MK;Bonham, MP;Chen, M;Cheng, F;Chikowore, T;Chivers, SC;Clemmensen, C;Dabelea, D;Dawed, AY;Deutsch, AJ;Dickens, LT;DiMeglio, LA;Dudenhöffer-Pfeifer, M;Evans-Molina, C;Fernández-Balsells, MM;Fitipaldi, H;Fitzpatrick, SL;Gitelman, SE;Goodarzi, MO;Grieger, JA;Guasch-Ferré, M;Habibi, N;Hansen, T;Huang, C;Harris-Kawano, A;Ismail, HM;Hoag, B;Jones, AG;Koivula, RW;Leong, A;Leung, GKW;Libman, IM;Liu, K;Lowe, WL;Morton, RW;Motala, AA;Onengut-Gumuscu, S;Pankow, JS;Pathirana, M;Pazmino, S;Perez, D;Petrie, JR;Powe, CE;Quinteros, A;Jain, R;Ray, D;Ried-Larsen, M;Saeed, Z;Santhakumar, V;Kanbour, S;Sarkar, S;Monaco, GSF;Scholtens, DM;Selvin, E;Sheu, WH-H;Stanislawski, MA;Steenackers, N;Steck, AK;Stefan, N;Støy, J;Taylor, R;Tye, SC;Ukke, GG;Urazbayeva, M;Van der Schueren, B;Vatier, C;Wentworth, JM;Hannah, W;White, SL;Yu, G;Zhang, Y;Zhou, SJ;Beltrand, J;Polak, M;Aukrust, I;de Franco, E;Flanagan, SE;Maloney, KA;McGovern, A;Molnes, J;Nakabuye, M;Njølstad, PR;Pomares-Millan, H;Provenzano, M;Saint-Martin, C;Zhang, C;Zhu, Y;Auh, S;de Souza, R;Fawcett, AJ;Gruber, C;Mekonnen, EG;Mixter, E;Sherifali, D;Eckel, RH;Nolan, JJ;Philipson, LH;Brown, RJ;Billings, LK;Boyle, K;Costacou, T;Dennis, JM;Florez, JC;Gloyn, AL;Gomez, MF;Gottlieb, PA;Greeley, SAW;Griffin, K;Hattersley, AT;Hirsch, IB;Hivert, M-F;Hood, KK;Josefson, JL;Kwak, SH;Laffel, LM;Lim, SS;Loos, RJF;Ma, RCW;Mathieu, C;Mathioudakis, N;Meigs, JB;Misra, S;Mohan, V;Murphy, R;Oram, R;Owen, KR;Ozanne, SE;Pearson, ER;Perng, W;Pollin, TI;Pop-Busui, R;Pratley, RE;Redman, LM;Reynolds, RM;Semple, RK;Sherr, JL;Sims, EK;Sweeting, A;Tuomi, T;Udler, MS;Vesco, KK;Vilsbøll, T;Wagner, R;Rich, SS;Franks, PW;Ada/Easd, P
    Contributors: Institute of Molecular and Genomic Medicine
    Abstract: Type 1 diabetes (T1D) results from immune-mediated destruction of insulin-producing beta cells. Prevention efforts have focused on immune modulation and supporting beta cell health before or around diagnosis; however, heterogeneity in disease progression and therapy response has limited translation to clinical practice, highlighting the need for precision medicine approaches to T1D disease modification.
    Date: 2023-10-05
    Relation: Communications Medicine. 2023 Oct 05;3:Article number 130.
    Link to: https://doi.org/10.1038/s43856-023-00357-y
    Cited Times(Scopus): https://www.scopus.com/inward/record.url?partnerID=HzOxMe3b&scp=85173487646
    Appears in Collections:[許惠恒] 期刊論文

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